Hepatic fibrogenesis can be reduced by suppressing aHSCs or returning to a quiescent phenotype.[20] To determine whether Mφtgmif resolved liver fibrosis by influencing HSCs, liver sections were stained with aHSC markers, including alpha‐smooth muscle actin (α‐SMA), TGF‐β, and collagen I (COL‐1). The gene discussed is ACTA1; the disease is Hepatic fibrosis.