Combined with the data from the animal sera, it is reasonable to suggest that OA can control obesity by regulating PPAR\documentclass[12pt]{minimal}\usepackage{amsmath}\usepackage{wasysym}\usepackage{amsfonts}\usepackage{amssymb}\usepackage{amsbsy}\usepackage{mathrsfs}\usepackage{upgreek}\setlength{\oddsidemargin}{-69pt}\begin{document}$$\gamma$$\end{document}γ/SREBP1 to control lipid synthesis. The gene discussed is SREBF1; the disease is obesity disorder.