By promoting expression of CPT1A, long-chain acyl-CoA dehydrogenase (LCAD) and fatty acid oxidation-related genes in CD8+ TILs, it maintains the number of CTLs and inhibits tumour progression in a lung cancer transplanted tumour model [59], which is consistent with the research of Saibil et al. [60]. The gene discussed is ACADL; the disease is lung cancer.