BAX and pancreatic neoplasm: Calycosin (25–200 μM) prevented the growth and viability of pancreatic cancer cells (PaCa‐2, PANC1) by triggering p21Waf1/Cip1‐induced cell cycle arrest at S phase and caspase‐dependent apoptosis via caspase‐3,8,9 activation, elevating Bax expression while lowering Bcl‐2, consequently increasing the ratio of Bcl‐2 to Bax.81