AKT1 and uterine cervix neoplasm: The phosphatidylinositol 3-kinase/Akt (PI3K/AKT) pathway has the capacity to modulate the expression of key oncogenes (e.g. HCCR-1) and tumor suppressor genes (e.g. p53) 91 and elicit the transition between epithelial and stromal tissues 92, thereby serving as a facilitator in the onset and advancement of cervical tumors.