In adult OcnGK−/− mice, the hyperglycemia was also of similar severity whether their mothers were OcnGK+/− or OcnGK−/− (Fig. 2A,B), verifying that a single Ocn allele in mothers cannot overcome the deleterious consequences on glucose homeostasis in offspring resulting from the absence of Ocn expression in embryos. Here, BGLAP is linked to Hyperglycemia.