In another study by Xia et al., it was revealed that the overexpression of the mitochondrial protein YME1 Like 1 (YME1L) promoted Akt-mTOR cascade activation in NSCLC cells, while the silencing or knockout of YME1L resulted in a reduction in signaling [47]. This evidence concerns the gene YME1L1 and non-small cell lung carcinoma.