Identifying the function of the Cdr1as/miR-195 axis in intestinal epithelium is of particular importance from a clinical point of view because human intestinal mucosa with injury/erosions and inflammation from patients with IBD and sepsis exhibited increased levels of Cdr1as that were associated with the deregulated expression of many miRNAs, including miR-195, as reported previously (31, 52). The gene discussed is LINC00632; the disease is inflammatory bowel disease.