Coagulation disorders such as elevated dimers are associated with direct damage to multiorgan endothelial cells by the COVID-19 virus and the release of inflammatory factors such as IL6 caused by infection, leading to a hypercoagulable state, which can lead to an increased risk of thrombosis in the venous and arterial systems, as well as in the microvascular system of vital organs such as the lungs and kidneys [18]. This evidence concerns the gene IL6 and blood coagulation disease.