In conclusion, miR-152 is lowly expressed in cervical cancer tissues, and its overexpression suppresses the proliferation, migration and infiltration and reduces the DDP resistance of cervical cancer cells by inhibiting the expressions of related proteins of the ERBB3/Akt/c-myc and ERBB3/Akt/Snail pathways and EMT. Here, AKT1 is linked to cervical cancer.