IFNL3 and hepatitis B virus infection: Some SNPs have been described as influencing factors in the clinical course of viralhepatitis B and C. For example, evidence has been presented that links some allelesof SNP rs8099917 located on chromosome 19 in the exon of the Interferon lambda 3(IFNL3) gene near the non-coding region [6,7], associated to predisposition ofchronic hepatitis B virus (HBV) infection susceptibility; to the level of fibrosisin histological findings in hepatitis C virus (HCV) infection; and the response totreatment with α-PEG-IFN/RBV in hepatitis C [8, 9].