The results revealed that LUAD is mainly enriched in the extrinsic component of membrane, lymphocyte differentiation, cell morphogenesis, I-κB kinase/NF-κB signalling, and immune system process (Figs. 6A and 6B), as well as KEGG pathway terms such as lysosome, rheumatoid arthritis, arrhythmogenic right ventricular cardiomyopathy, adherens junction, and Staphylococcus aureus infection (Figs. 6C and 6D). This evidence concerns the gene NFKB1 and Arrhythmogenic right ventricular dysplasia.