NFKB1 and arrhythmogenic right ventricular cardiomyopathy: The results revealed that LUAD is mainly enriched in the extrinsic component of membrane, lymphocyte differentiation, cell morphogenesis, I-κB kinase/NF-κB signalling, and immune system process (Figs. 6A and 6B), as well as KEGG pathway terms such as lysosome, rheumatoid arthritis, arrhythmogenic right ventricular cardiomyopathy, adherens junction, and Staphylococcus aureus infection (Figs. 6C and 6D).