In an RNA-seq analysis of oral mucosal ulcers and diabetic foot ulcer tissues, one investigator found that keratins associated with trauma activation (KRT6, KRT16, and KRT17) and keratins associated with cell differentiation (KRT1, KRT2, and KRT10) were upregulated in oral mucosal ulcer tissues to promote ulcer wound repair. Here, KRT1 is linked to diabetic foot.