FAAH and colorectal carcinoma: Moreover, it was shown that in azoxymethane (AOM)-induced CRC, the application of cannabinoid agonist HU210 and inhibitor of endogenous cannabinoid-hydrolyzing enzyme (fatty acid amide hydrolase, FAAH) results in the decreased formation of precancerous aberrant crypt foci [48] and lesions in xenograft models [49].