The majority of the genes involved in Alzheimer’s disease increased following SARS-CoV-2 infection and are also involved in neuroinflammation (CXCL8, EGFR, IL-17, IL-18, IL-6R, and LGALS3), while others regulate neuronal apoptosis (KLF4), tau phosphorylation (FKBP5), or glial cell activation (GFAP, EGFR) [34]. Here, EGFR is linked to early-onset autosomal dominant Alzheimer disease.