Only a small proportion of HF-dysregulated genes reached the control levels following LVAD implantation; however, different mRNAs of pro-inflammatory pathways are dysregulated and included (C/EBPβ [enhancer-binding protein beta], NFKBIA [NFKB inhibitor alpha], CXCL12 [C-X-C motif chemokine ligand 12], CCL2 [C-C motif chemokine ligand 2], and CD14). The gene discussed is NFKBIA; the disease is hydrops fetalis.