The study observed that the deletion variant of the 94ins/delATTG polymorphism in the NF-κB1 gene’s promoter region, whether homozygous (DD) or heterozygous (WD), was linked to an increased risk in CRC among Swedish patients, including those with both unselected and sporadic forms of the disease [36,40]. This evidence concerns the gene NFKB1 and colorectal carcinoma.