CREB1 and endometriosis: Guo and his colleagues claimed that chronic stress accelerates endometriosis development and is mediated by the activation of Adrenoceptor Beta 2 (ADRB2) and cyclic-AMP response binding protein (CREB) pathways, and that antagonism of ADRB2 signaling might be a new therapeutic avenue for the treatment of endometriosis [11,21].