Based on molecular characteristics, epithelial ovarian cancers can be subdivided into type I cancers (endometrioid, clear cell, mucinous, and low-grade serous carcinomas), which are rarely associated with a TP53 mutation, usually indolent, and present at a younger age, and type II carcinomas (high-grade serous carcinoma, undifferentiated carcinoma, and carcinosarcoma), which have unknown precursor lesions, frequently carry a TP53 mutation, and are aggressive in nature [13]. This evidence concerns the gene TP53 and serous adenocarcinoma.