IFNG and extranodal nasal NK/T cell lymphoma: For example, in nasal natural killer/T-cell lymphoma (NKTCL), which is commonly linked to Epstein–Barr virus (EBV), miR-142-3p is downregulated and IFNγ production is inhibited, thereby promoting lymphoma transformation with increased oncogenic BCL6 and IL1A cytokine expression [84,85].