NFKB1 and endometritis: The NF-κB signaling pathway is a key pathway that regulates cellular redox status and anti-stress and anti-inflammatory responses [42], miR-93-5p promotes apoptosis of granulosa cells in polycystic ovary syndrome through the NF-κB signaling pathway [43], and GNRB1 inhibits lipopolysaccharide-induced endometritis in mice by suppressing the NF-κB signaling pathway [44].