As a result of weak GMA intervention, the expression of TLR4, TRAF6, NF-κB, and TNF-α was significantly decreased, the expression of MyD88 and MCP-1 was increased, the inflammatory response was blunted, the area of myocardial infarction was reduced, and cardiac function was improved. The gene discussed is MYD88; the disease is myocardial infarction.