Although a wealth of studies have accumulated on the immunopathogenesis of NASH, there has been an increase in IL-6 and IFN-γ8 due to the over-activation of hepatic immune cells, accompanied by the downregulation of IL-10.25 Interestingly, in our study, we observed a statistically significant increase in serum IL-10 levels and non-statistical decreases in serum IFN-γ and IL-6 levels in the NASH group. The gene discussed is IFNG; the disease is metabolic dysfunction-associated steatohepatitis.