Genetic deletion or pharmacological inhibition of neutral endopeptidase exacerbated lung injury and elevated pancreatic myeloperoxidase both in CER-AP and in choline-deficient ethionine-supplemented diet-induced AP (CDE-AP), a severe necrotizing model caused by deranged amino acid metabolism in young and female mice (Maa et al., 2000; Koh et al., 2011b). Here, MPO is linked to alkaline phosphatase measurement.