Furthermore, the pathways linked to GX0101-induced tumors in both the 30 dpi and 45 dpi experimental groups included mTOR, MAPK, cAMP, ECM-receptor interaction, Chemokine, JAK-STAT, MicroRNAs in cancer, ErbB, pathways in cancer, Rap1, VEGF, Ras, Wnt, Prion diseases, and Hippo pathway. Here, SOAT1 is linked to prion disease.