Previous studies have demonstrated that LDLR is also a receptor for VSV,23 Hepatitis C Virus (HCV),45 and human rhinovirus 2 (HRV2).46 However, LDLR-deficiency did not abolish VSV,23 HCV47,48 and HRV2 infection,46 suggesting that LDLR is redundant for entry of these viruses. The gene discussed is LDLR; the disease is infection.