A mouse model of DMD lacking all neuronal Dystrophin isoforms (mdx) exhibits defects in CCK+/CB1R+ IN synapse development and abnormal innervation in the hippocampus, resembling the innervation pattern we observed in Emx1Cre:Dag1 cKO and Emx1Cre:Pomt2 cKO mice in this study (Krasowska et al., 2014). Here, CCK is linked to Duchenne muscular dystrophy.