In addition, although the IFN-β treatment and Dex treatment groups both showed reduced respiratory system resistance and airway resistance in virus-induced asthma exacerbation mice, IFN-β treatment was significantly more effective in preserving the respiratory system compliance and tissue damping and elasticity (Fig. 8c), which indicates that IFN-β treatment not only has an effect on the airways, but also has a good effect of improvement on AHR caused by tissue damage. This evidence concerns the gene IFNB1 and asthma.