Additionally, lymphotoxin derived from cancer cells can induce chemokine expression in stromal fibroblasts via the LTBR-NF-κB signaling pathway, while the lymphotoxin-LTBR and CXCL11-CXCR3 signaling pathways serve as therapeutic targets in ovarian cancer [26]. This evidence concerns the gene CXCL11 and ovarian carcinoma.