His research on the pathogenesis of psoriasis has made significant contributions to Langerhans cells, inflammatory responses mediated by inflammasomes like Nod‐like receptor pyrin domain containing 3 (NLRP3) and Absent in melanoma 2 (AIM2)21, 22; the polarization effect of miR‐381‐3p on Th1 and Th1723; the differentiation effect of glycolysis metabolism on Th17 and Th124; adipocytes and C1q/tumor necrosis factor‐related protein 3 (CTRP3) mediated Lysosome‐associated membrane protein 1 (LAMP1)/ Signal transducers and activators of transcription (STAT3) axis25 in psoriasis. The gene discussed is STAT3; the disease is psoriasis.