It is generally accepted that apart from blood incompatibilities, breastfeeding, prematurity and family history of NHB [10], liver dysfunction such as intrahepatic cholestasis of pregnancy (ICP) in mothers during pregnancy leading to disturbances of alanine aminotransferase (ALT), aspartate aminotransferase (AST), and total bile acid (TBA) markers are also risk factors for the development of NHB [11]. This evidence concerns the gene GPT and Intrahepatic cholestasis of pregnancy.