In summary, we consider that after MYCT1 overexpression, it may interact with some transcription factors or proteins, causing cell cycle arrest, inactivating oncogenes or activating tumor suppressor genes of the DLBCL cell line itself, and losing the previously stable immune escape function, which will lead to increased apoptosis and inhibit the proliferation of lymphoma cells. Here, MYCT1 is linked to diffuse large B-cell lymphoma.