Knockout of EEF1A2 reduced cell proliferation and migration, and increased apoptosis in AML-193 and Kasumi-1, which was rescued by ectopic expression of EEF1A2. Mechanistically they found increased dimethylation of eEF1A at lysine 55 in AML-193, Kasumi-1, and KG-1. The gene discussed is EEF1A2; the disease is acute myeloid leukemia.