In addition, LOXL1-AS1 sponges miR-515-5p to inhibit its binding with STAT3, thereby increasing the expression of STAT3, forming a LOXL1-AS1/miR-515-5p/STAT3 positive feedback loop, and promoting cell proliferation and migration in atherosclerosis (AS) [32]. This evidence concerns the gene LOXL1 and atherosclerosis.