ATP10A and obesity disorder: However, we found no evidence for increased obesity or metabolic defects in female Atp10A+/- heterozygous mice inheriting the KO allele paternally or maternally (Supplemental Fig. 3), nor did the Atp10A−/− homozygous mice display alterations to the development of diet-induced obesity or defects in glucose metabolism (Figs. 1, 2).