They suggested that investigation of the role of N‐terminal splicing in other primary tauopathies associated with different pathologies – such as Pick's disease, primary age‐related tauopathy (PART), and chronic traumatic encephalopathy (CTE) – would help determine whether such diverse disorders also exhibit loss of MAPT exon 2 and 10 splicing coordination. The gene discussed is MAPT; the disease is Pick disease.