found that DJ-1 can not only induce EMT transcription factor (TF) to activate the epithelial-mesenchymal transition (EMT) program through the TrkC/DJ-1/STAT3 signaling pathway, but also obtain anticancer drug resistance through TrkC-mediated inhibition of DJ-1 degradation30, and studies and other studies have shown that DJ-1 also receives the regulation of different miRNAs upstream in the form of target genes, which has an impact on tumor growth and drug sensitivity XL31. Here, NTRK3 is linked to neoplasm.