Previous studies have shown that overexpression of Glut1 in VSMCs promotes chemokine CCL2 and monocyte chemoattractant protein-1 (MCP-1) expression and monocyte recruitment, resulting in excessive inflammatory reactions and accelerating atherosclerosis (Adhikari et al. 2011; Hall et al. 2001; Kaiser et al. 1993; Peiró et al. 2016; Vesely et al. 2009; Wall et al. 2018). The gene discussed is SLC2A1; the disease is atherosclerosis.