Saroglitazar, a different dual PPARα/γ agonist, improved steatosis, lobular inflammation, hepatocellular ballooning, and fibrosis levels in an animal model of NASH 109 and it was found to ameliorate ALT, liver fat content, insulin resistance, and atherogenic dyslipidemia in NASH patients, along with positive histological indications110. The gene discussed is GPT; the disease is metabolic dysfunction-associated steatohepatitis.