In mice with adriamycin nephrosis (experimental FSGS, induced at 3–4‐months and followed for 4 weeks), SLK deletion exacerbates albuminuria (~5‐fold greater in SLK KO mice compared to control), and loss of podocytes (Woychyshyn et al., 2020). This evidence concerns the gene SLK and focal segmental glomerulosclerosis.