IGKV2D-24 and Alzheimer disease: Given the links between oAβ42/α7β2‐nAChR interactions on basal forebrain cholinergic neurons to critical events in early AD, and the more restricted distribution of α7β2‐ versus α7‐only‐nAChR,17, 19, 20, 21, 23 α7β2‐nAChR could be an exceptionally significant pharmacotherapeutic target.