The mechanisms of cuproptosis involve the gene models of copper homeostasis dysregulation, including cuproptosis regulator MTF1 (metal-regulatory transcription factor 1), the copper exporters ATP7A (ATPase copper-transporting alpha) and ATP7B (ATPase copper-transporting beta), which are encoded by genes that are mutated in the copper dysregulation syndrome Wilson’s disease [58, 59]. The gene discussed is MTF1; the disease is Wilson disease.