ACTA1 and pancreatic ductal adenocarcinoma: This categorisation is exemplified by the induction of IL‐6+ CAFs (iCAF) and α‐SMA+ CAFs (myCAF) from quiescent pancreatic stellate cells in pancreatic ductal adenocarcinoma through IL‐1 and tumour growth factor‐beta (TGF‐β) stimulation.59, 60