cGAS promotes DNA damage repair by activating the STING‐TBK1 pathway.[24] Protein mass spectrometry was used to evaluate protein changes in PTCL cell lines (MT‐4 and Hut78) after G150 treatment to explore the applicability of this mechanism to PTCL. The gene discussed is TBK1; the disease is mature T-cell and NK-cell non-Hodgkin lymphoma.