The exact mechanisms underlying renal injury caused by SARS-CoV-2 are not yet clear, but studies have suggested that direct viral effects, Renin-Angiotensin-Aldosterone System (RAAS) imbalance, cytokine storms, organ crosstalk, hypercoagulability, complement activation, downregulation of ACE2, microvascular injury, COVID-19-associated prothrombotic state, and other factors may contribute to kidney damage (Gabarre et al., 2020; Zheng et al., 2021; Meng, 2022). This evidence concerns the gene ACE2 and Nephropathy.