IFNG and infection: We suggest that the mechanistic details triggered by these parasites, especially in ML, induce a greater expression of micro RNAs that modulate pathways of initial host–pathogen interaction and that, finally, provide parasites with the ability to multiply and escape cells after infection by interfering mechanisms such as phagocytosis, secretion of chemokines and prostaglandins, and the inhibition of IFN-γ inducible pathways.