EOC patients showed higher levels of proinflammatory cytokines (IL-6, TNF-α, and IL-12), regulatory cytokines (IL-10), and chemokines (CXCL-9 and CXCL-10), which corroborated this environmental proinflammatory/regulatory mechanism for the development of ovarian cancer. The gene discussed is CXCL10; the disease is ovarian carcinoma.