Similar observations have been reported in studies in cutaneous basal cell carcinoma, showing that K17 levels directly correlate with changes in the expression of inflammatory T-helper cytokines [8,18], and colocalization of K17 with key cytokines, including CXCR3, CXCL10, and CXCL11 [19], as well as in a cervical cancer mouse model where K17 high expressing lesions had increased transcript levels of pro-inflammatory cytokines, including interferon gamma, CXCL9, CXCL10 and CXCL11 [20]. This evidence concerns the gene KRT17 and cervical cancer.