Unfortunately, in high-CIN cancers, cGAS-STING promotes motility and metastasis [155,159], most likely due to the aforementioned connection between STING activation and upregulation of NF-κB, which subsequently elevates the expression of transcription factors associated with epithelial-mesenchymal transition (EMT). The gene discussed is NFKB1; the disease is cervical squamous intraepithelial neoplasia.