This response was not observed when gene expression of iNOS was determined using total lung homogenates, but as seen in Figure 2D, the percentage of lung surface affected by pneumonia was lower in the AdTNF group, along with a lower bacterial load (Figure 2C), so inflammatory consolidation in the lungs of AdTNF mice is lower and pro-inflammatory molecules are more highly expressed in these areas where bacteria are more numerous. This evidence concerns the gene NOS2 and pneumonia.